training with type two diabetes

🔥+ training with type two diabetes 09 Jul 2020 HbA1c Control ≤ 9 for Members with Diabetes : The percentage of members 18–75 years of age with diabetes (Type 1 & Type 2) whose most recent HbA1c level (performed ... To assist health care professionals with the transition to ICD-10 codes, the ... The code is structured into 3 distinct Levels, including provider type, ...

training with type two diabetes ... about diabetes mellitus type 2 (also referred to as diabetes mellitus 2) and the ... current and emerging treatment of chronic kidney disease in type 2 diabetes.

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training with type two diabetes fruits (⭐️ teenager) | training with type two diabetes winehow to training with type two diabetes for insulin resistance and islet cell dysfunction (neither completely)

Patients with Type 2 diabetes are about half as responsive to insulin as non-diabetic individuals matched for age, sex and weight
-Resistance to insulin increases as weight increases in both diabetics and non-diabetics
-All obese individuals are insulin resistant to some degree and many non-diabetic conditions are associated with insulin resistance
-Insulin receptors are structurally normal and binds insulin normally in Type 2 diabetes, so that the decrease in insulin action lies at a post receptor site.

Patients with Type 2 diabetes have less than 50 % of the number of islets as age matched non-diabetics. Also, islets in patients with type 2 diabetes accumulate a proteinaceous material, amylin, that may be toxic to the cells.
Preventing pre-diabetes from becoming diabetes: 5% weight reduction, 30 minutes of exercise a day, dietary changes (less fat, less sat fat, more fiber)

relief of acute symptoms and prevention of the 1 last update 09 Jul 2020 microvascular complications through glucose control

also treatment of hypertension, hypercholesterolemia, and obesity

diet: limit simple sugars and sat fats
exercise: mix of aerobic and resistance is most effective for lowering blood sugar
education
pharmacologyPreventing pre-diabetes from becoming diabetes: 5% weight reduction, 30 minutes of exercise a day, dietary changes (less fat, less sat fat, more fiber)

relief of acute symptoms and prevention of microvascular complications through glucose control

also treatment of hypertension, hypercholesterolemia, and obesity

diet: limit simple sugars and sat fats
exercise: mix of aerobic and resistance is most effective for lowering blood sugar
education
pharmacology
type II is most common worldwide

overall US prevalence: 6% (but probably higher as many undiagnosed)
over the age of 50: 16%
14-15% of people have pre-diabetes

Ethnicity
-estimated lifetime risk for individuals born in the US in the year 2000, between 30 to 40%
-higher in African Americans
-even higher in Hispanic Americans
-high in native americans

Major risk factors: obesity, family history, prior glucose intolerance, prior gestational diabetes

Obesity
-responsible for the geographic distribution we see in the US.
-Central obesity

Family history
-100% concordance with identical twin (usually get it when they hit the same weight, not so much about age). This is v different that type I.
-this is a many gene disease. the genes are broadly either related to islet cells and weight. Aren''t respond normally to IV stimulus because they have islet cell dysfunction, don''s not that much compared to muscle
-also regulates free fatty acid output
-no insulin, too much fatty acid in the blood
-high levels of free fatty acid enhance or may even causes insulin resistance in the liver
Adiponectin
-hormone made by fat cells that makes you more sensitive to insulin (muscle tissues)
-less in Diabetes compared to weight matched non-diabetic control
-the phase 2 for this as a treatment was stopped

Leptin
-increases with increasing fat storage and inhibits appetite
-obese individuals must have some levels of leptin resistance
-will likely be increased in prediabetes and diabetes

GLP1 aka incretin
-secreted by jejunum in response to eating, enteroendocrine cells (secretory granules)
-increases insulin release
-decreases glucagon release
- slows stomach emptying/inhibits appetite
-don''s the sensor, the mutation makes it slower, takes a higher level of glucose to kick the islet cell into action and make insulin, fewer complications

Pima Indians
-no diabetes or diabetes before being put on the reservation
-now 90% obese and greater than 40% have diabetes

Adolescent type 2 diabetes
-4/1000 kids in the US, higher in native populations
1)Increased insulin resistance of puberty added to insulin resistance of obesity and genetic background.
2) Fetal factors: poor maternal metabolic control in diabetic mothers
3) fetal environment may have long term metabolic consequences.
4)Environmental factors - namely diet composition and exercise are thought to underlie increased prevalence
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